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glutathione and breast cancer risk An integrative multi-omics analysis leveraging Mendelian randomization and subsequent experimental validation prioritizes glutathione S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in
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glutathione and breast cancer risk An integrative multi-omics analysis leveraging Mendelian randomization and subsequent experimental validation prioritizes glutathione S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in
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Involvement of glutathione peroxidases in the occurrence and development of breast cancers Journal of Translational Medicine Springer Nature Link Frontiers Glutathione responsive and exhausting metal nanomedicines for robust synergistic cancer therapy Role of Glutathione in Cancer: From Mechanisms to Therapies International Journal of Oncology
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glutathione and breast cancer risk An integrative multi-omics analysis leveraging Mendelian randomization and subsequent experimental validation prioritizes glutathione S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in
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